KV9.2 Polyclonal Antibody

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¥680.00

市场价:¥880.00

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产品规格:
  • 50μL
  • 100μL
货号:

FAB2614716

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商品描述
  • 靶点-Target

    KV9.2

  • 反应性

    Human,Mouse,Rat

  • 应用

    WB,IHC

  • 宿主物种-Host Species

    Rabbit

  • 同种型-Isotype

    IgG

  • 修饰-Modification

    Unmodified

  • MW(Observed)

    54kD

  • 推荐稀释比

    WB 1:500-2000;IHC 1:50-300

  • 组成

    Liquid in PBS containing 50% glycerol, 0.5% BSA and 0.02% sodium azide.

  • 纯化工艺

    The antibody was affinity-purified from rabbit antiserum by affinity-chromatography using epitope-specific immunogen.

  • 储存

    -15°C to -25°C/1 year(Do not lower than -25°C)

  • 浓度

    1 mg/ml

  • 克隆性

    Polyclonal

  • 免疫原

    The antiserum was produced against synthesized peptide derived from human KCNS2. AA range:197-246

  • 特异性

    KV9.2 Polyclonal Antibody detects endogenous levels of KV9.2 protein.

  • 基因名称

    KCNS2

  • 蛋白名称

    Potassium voltage-gated channel subfamily S member 2

  • 别名

    KCNS2;KIAA1144;Potassium voltage-gated channel subfamily S member 2;Delayed-rectifier K;+;channel alpha subunit 2;Voltage-gated potassium channel subunit Kv9.2

  • Organism-1

    Human

  • 基因ID-1

    3788

  • SwissProt-1

    Q9ULS6

  • Organism-2

    Mouse

  • 基因ID-2

    16539

  • SwissProt-2

    O35174

  • Organism-3

    Rat

  • 基因ID-3

    66022

  • SwissProt-3

    Q9ER26

  • 背景

    domain:The segment S4 is probably the voltage-sensor and is characterized by a series of positively charged amino acids at every third position.,function:Potassium channel subunit. Modulates channel activity and reduces the ion flow.,similarity:Belongs to the potassium channel family. S subfamily.,subcellular location:May not reach the plasma membrane but remain in an intracellular compartment in the absence of KCNB1.,subunit:Heteromultimer with KCNB1 and with KCNB2. Does not form homomultimers. Might also bind to other channel proteins.,

  • 细胞定位

    Cell membrane ; Multi-pass membrane protein . May not reach the plasma membrane but remain in an intracellular compartment in the absence of KCNB1 or KCNB2. .